Inquisitores ad University of California, San Francisco et iudae filii 's Research Tennessee hospitalis medicinae mysterium solvitur enim decenniis, et inventa sunt de geneticae mutationes, ut par faciam rem domesticam nouerant morbo et sanguinem leukemia. Hoc studio fundatur in analysis de DNA XVI V siblings per familias, et liberos simulque ostendens, quod aliqua mutationes in hæreditatem recuperandam suis, et inventus est alius quidam geneticae venalicium quod vitare auxilium medicus Psidium et medullis ossa periculo transplantatio.
De quaestionibus quo morbus potest reduci magis quam XXX annos, cum oncologist Kevin adunatus, MD, de University of California, San Francisco et sociis occurrit aliquot familias, multis ex quo potest habere humilis sanguinem cellula Comites (abnormes myelodysplastic syndrome) or Sign MDS) et subtiliter myeloid leukemia (AML), a gravibus atque exitiale sanguinem cancer. His aegris habent solito loco Homo VII duae, una quae vocatur Homo VII.
The data shows that mutations in the genes SAMD9 and SAMD9L located on chromosome 7 are closely related to single chromosome 7 syndrome, but many healthy siblings and patients’ parents also carry these mutations without any symptoms. Researchers have shown that patients who do have symptoms of MDS and AML also have a specific secondary gene mutation, which can drive a more serious disease, and patients without these additional mutations often never experience any symptoms and may develop blood. The count is low but most can recover on their own without treatment.
Paris chromosomatum habet geneticae mutationes in VII vitia frequentiora longioraque sunt in aegris cum MDS et AML, et malignus tumores ex uno chromosomate consociata sunt, in VII deploratae et pauper, non ut bene Respondeo dicendum existentium therapies. VII super genes Homo plus et DCCCLX, erit interesting intelligere, et per partes SAMD7 SAMD7L in non-oeconomicam et MDS AML et quomodo illi inter se occurrunt apud alios paris chromosomatum habet VII super genes.
